By Michelle M. Le Beau, Janet D. Rowley (auth.), Harry Harris, Kurt Hirschhorn (eds.)
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This publication constitutes the refereed complaints of the seventh ecu convention on Genetic Programming, EuroGP 2004, held in Coimbra, Portugal, in April 2004. The 38 revised papers awarded have been conscientiously reviewed and chosen from sixty one submissions. The papers take care of quite a few foundational and methodological matters in addition to with complicated functions in parts like engineering, desktop technological know-how, language figuring out, bioinformatics, and layout.
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Extra resources for Advances in Human Genetics 15
An exception to this correlation, namely, a Burkitt tumor with a t(8;22) whose cells produced IgMK, raises the question of whether the translocation event is intimately associated with the usual DNA rearrangement that is required to produce a functional immunoglobulin gene, or whether the two are independent. These data must be considered in the context of the observation by a number of investigators that the functional immunoglobulin gene locus is not the one involved in the translocation. However, the immunoglobulin gene on the 14q + is transcribed, and thus mRNA from this gene can be detected.
All three lines with a t(2;8) expressed Klight chains, and all five cell lines with a t(8;22) expressed A light chains. Of 17 cell lines with t(8;14), nine were K- and eight were A-producers. An exception to this correlation, namely, a Burkitt tumor with a t(8;22) whose cells produced IgMK, raises the question of whether the translocation event is intimately associated with the usual DNA rearrangement that is required to produce a functional immunoglobulin gene, or whether the two are independent.
The abnormal chromosomes 9 and 11 are on the right in each pair. (e) t(6;9)(p23;q34). The abnormal chromosome 6 is on the right of pair 6. The rearranged chromosome 9 is on the left of pair 9. (0 inv(3)(q21q26). Inverted chromosome 3 is on the right. The abnormality initially appeared to be restricted to patients with a diagnosis of M2leukemia (acute myeloblastic leukemia with maturation) according to the FAB classification. However, 7% (3 of 44) of patients analyzed at the Fourth Workshop whose cells had a t(8;21) and adequate bone marrow material available for cytological review had a diagnosis of M4leukemia (acute myelomonocytic leukemia).
Advances in Human Genetics 15 by Michelle M. Le Beau, Janet D. Rowley (auth.), Harry Harris, Kurt Hirschhorn (eds.)